Human Genome Epidemiology Literature Finder
|
Records 1 - 6 (of 6 Records) |
| Query Trace: Graves Disease and CD28[original query] |
|---|
| CTLA-4 gene polymorphism at position 49 in exon 1 reduces the inhibitory function of CTLA-4 and contributes to the pathogenesis of Graves' disease. Journal of immunology (Baltimore, Md. : 1950) 2000 Dec 165 (11): 6606-11. Kouki T, Sawai Y, Gardine C A, Fisfalen M E, Alegre M L, DeGroot L |
| Coeliac disease: investigation of proposed causal variants in the CTLA4 gene region. European journal of immunogenetics : official journal of the British Society for Histocompatibility and Immunogenetics 2003 Dec 30 (6): 427-32. King A L, Moodie S J, Fraser J S, Curtis D, Reid E, Dearlove A M, Ciclitira P |
| Evidence for unique association signals in SLE at the CD28-CTLA4-ICOS locus in a family-based study. Human molecular genetics 2006 Nov 15 (21): 3195-205. Cunninghame Graham D S, Wong A K, McHugh N J, Whittaker J C, Vyse Timothy |
| CD28/CTLA-4/ICOS haplotypes confers susceptibility to Graves' disease and modulates clinical phenotype of disease. Endocrine 2016 Sep . Pawlak-Adamska Edyta, Frydecka Irena, Bolanowski Marek, Tomkiewicz Anna, Jonkisz Anna, Karabon Lidia, Partyka Anna, Nowak Oskar, Szalinski Marek, Daroszewski Jac |
| Association of single nucleotide polymorphism in CD28(C/T-I3 + 17) and CD40 (C/T-1) genes with the Graves' disease. JPMA. The Journal of the Pakistan Medical Association 2018 Jan 68 (1): 3-9. Mustafa Saima, Fatima Hira, Fatima Sadia, Khosa Tafheem, Akbar Atif, Shaikh Rehan Sadiq, Iqbal Furh |
| Finding the Common Single-Nucleotide Polymorphisms in Three Autoimmune Diseases and Exploring Their Bio-Function by Using a Reporter Assay. Biomedicines 2023 9 11 (9): . Yen-Chang Chu, Kuang-Hui Yu, Wei-Tzu Lin, Wei-Ting Wang, Ding-Ping Ch |
- Page last reviewed:Feb 1, 2024
- Content source: